Contrary to popular opinion or the rules set down by the authorities riding a bicycle facing traffic is much safer than riding with traffic in the opinion of the author. When riding against traffic, a person can see what is coming and get off the highway when traffic gets too close.
There should also be proper lights, reflectors, bells or hors, etc. The NJ law states that a helmet should be worn by anyone 17 or younger. They should be in good condition and properly inflated. Brakes should be properly lubricated and be checked periodically for safety adjustments.
Apparel worn while riding can be very important. Avoid dark clothing. Reflective clothing could be very valuable. Lugs, chains, bolts, seats, handle bars, etc. should be checked periodically for safety and comfort. The help of a professional should be sought out during questionable adjustments and upkeep.
Per William Shakespeare “Neither a borrower or lender be!”
The worst is to lose a bike or have damage done to a bike is to lend someone your bike. People generally are less caring and/or protective of your property than they are of their own. I recommend you don’t be a "goody-two shoes "(Nice guy)
just to appease and/or be liked by the other person.
Since bicycles are a major source of transportation, in particular in Asbury Park, it is generally a good idea to avoid leaving the bike there.
Carrie Fisher, another celebrity death, common place, perhaps. We lost Robin Williams not that many years ago, there is a connection here. Both lived with (most people say suffered from) mental illness. Another “drug user” some might say, and yes have both of them admitted to using drugs. She came out with her illness before it became “popular” for celebrities. She also spoke about her addiction to drugs. Her autopsy results showed that that she was using them. Which is common for plenty of people who have mental illness to use them to cope with it.
Carrie Fisher got the conversation going about her mental illness and her addiction to drugs. Back when many people just hid it away from friends and family. She didn’t want those who live with it to feel shame because of it. She wanted an open conversation between those who live with it and those who don’t have mental illness. She wanted to break the stigma behind it, for there to be open discussion about it. The fact that drugs might have caused her death should not tarnish her as a person.
I found this on my personal Facebook page from a friend, and this was my response to it on her post:
“I understand, I used to think badly of all people using drugs until I got diagnosed with. I met plenty of people with drug and alcohol addictions. As I got to know them, as people, I lost that judgment, they were some really good people who were struggling with many issues and using and abusing was a way to cover up what felt. I know that not every drug abuser or alcoholic have mental illness, but a good majority are. So she did some drugs, like this stated she is a bad ass, to live with mental illness and get into movies or for the non-famous (us common folk) just getting up and go to work on a daily is too. I’m also including veterans who have fought for this country, mostly older vets and some of the ones retuning home now. The horrors that they saw and the never ending battles that they fight in their heads. Drugs is one way they cope with it as the government doesn’t do much for them. Don’t judge what you don’t understand. “
She was an influence early on when most people, not just in Hollywood, didn’t want to talk about mental illness. It was kept quiet and hardly ever bought to light. This should not diminish her part the she took into getting this to be talked about in the open about. I’m going leave you with that thought and leave you with some quotes of hers. Carrie Fisher you will always be “bad ass” not only as your role as the only big female in the original Star Wars trilogy but in what you have done in the mental health community. Rest in peace.
‘I’m mentally ill. I can say that. I am not ashamed of that. I survived that, I’m still surviving that, but being it on. Better me then you.” ~ December 2000 in an interview with Diane Sawyer.
“ One of the things that baffles me ( and there are quite a few) is how there can be so much lingering stigma with regards to mental illness, specifically bipolar disorder . In my opinion living with manic depression takes a tremendous amount of balls. Not unlike a tour of Afghanistan (though the bombs and bullets, in this case, come from inside.) At times, being bipolar can be an all-consuming challenge, requiring a lot of stamina and even more courage, so if you’re living with mental illness and functioning at all, it’s something to be proud of, not ashamed of. They should issue medals along with a steady stream of medication.” ~ From Wishful Drinking
The secret is out. The anesthetic drug, ketamine, can save lives for people with severe depressive disorders. It is being introduced in hospital emergency rooms as a rapid-acting antidepressant to treat people who have become suicidal. Patients can respond favorably in as little as one or two hours. By contrast, an antidepressant drug like Prozac can take weeks to work. In the hospital setting, ketamine is given intravenously (IV). The drug is infused as 0.5 mg/kg body weight over 40-45 minutes. This is a dose substantially lower than those used for anesthesia. It is also lower than what is used recreationally as “Special K’ to produce dissociative and psychotomimetic psychological effects.
Some of the earliest research for the use of ketamine in depression was conducted at the NIMH division of the National Institutes of Health. It was studied as a treatment for those people with Bipolar Disorder or Major Depressive Disorder who did not respond to traditional antidepressants. It was found that a substantial number of antidepressant non-responders responded to a single ketamine infusion within a few hours. Unfortunately, the therapeutic effect disappeared within a few days to a week. Eventually, it was determined that a patient can receive booster or maintenance doses once per month. Some people can go longer. Many patients are able to return to a life without depression.
Going for ketamine IV infusions once per month is inconvenient and expensive. Some doctors now offer ketamine as a nasal spray (intranasal) instead. Ketamine is very cheap. When prescribed for intranasal administration, one cannot simply have it filled at their local pharmacy. A compounding pharmacy is needed to create a liquid from the ketamine powder. As with IV ketamine, the dosage of intranasal ketamine must be well-controlled. Intranasal ketamine is often given as 40 mg doses, 5-7 days apart. However, it does happen that some people who do not respond to intranasal ketamine go on to respond to intravenous ketamine.
The dosing of ketamine needs to be precise. The reason for this is that too low a dosage doesn’t work, and too high a dosage produces dissociative states and even psychosis. Pharmaceutical companies have been trying to develop other drugs that do the same thing as ketamine on a biological level, but without the dissociative psychological side effects. Like ketamine, these drugs cause a blocking of a gate on neurons (nerve cells) that normally let in a neurotransmitter messenger molecule known as glutamate. This gate is controlled by a special receptor called the NMDA (N-Methyl-D-Aspartate) receptor. It was thought that blocking the NMDA receptor was critical to the mechanism by which ketamine worked to treat depression. The drugs in development are bound to be expensive. It turns out, though, that ketamine may not depend on NMDA at all. Ketamine is broken-down (metabolized) naturally in the liver into a substance known as HNK (hydroxynorketamine). HNK does not work on NMDA receptors at all. Instead, it causes an increase in the production of BDNF (brain-derived neurotrophic factor). BDNF stimulates the brain to grow new neurons (neurogenesis) and support those that already exist (neurotrophy). New neurons are formed in the hippocampus and cortical regions of the brain. These structures have been implicated in the development of depressive disorders. This yields a very important conclusion. HNK can be used without worrying about dissociative and psychotomimetic effects. Dosing does not need to be precise, and the drug can be given orally.
Ketamine doesn’t help everyone. It seems that a minority of people have a type of gene that causes BDNF to be manufactured less efficiently. Most people have the val66val gene that allows for the most efficient synthesis of BDNF. They respond almost completely to ketamine. However, others with the met66val gene respond less well, and those with met66met don’t respond at all. BDNF stimulates the growth of new neurons in the hippocampus structure of the brain. The hippocampus is responsible for regulating memory and emotions. In depression, the size of the hippocampus is reduced. When people respond to traditional antidepressants, there is an increase in neurogenesis there, and an increase in size.
For now, empirical evidence supports the use of ketamine in depressive disorders. Hopefully, its therapeutic biological mechanisms will be better understood with continued study, leading to better treatments for depression with fewer side effects. The ketamine metabolite, HNK, might be the ideal alternative to ketamine.
Blog posts are written by Shore House members and staff.